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Clinical evidence consistently demonstrates that GLP-1RAs exert cardioprotective effects, including a significant reduction in the risk of MACE, alleviation of heart failure-related symptoms, and delay in the progression of atherosclerotic lesions

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Insulin resistance in this group of patients results from defects at the molecular level, including impaired insulin receptor-related signaling pathways enhanced by obesity and its features: Excess visceral fat, chronic inflammation, and reactive oxygen species

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