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These platforms enable on-demand insulin or GLP-1 release triggered by elevated blood glucose levels, providing a closed-loop feedback mechanism that enhances therapeutic precision and reduces the risk of hypoglycemia [41]

Literature Review The existing literature demonstrates that GLP-1 receptor activation produces anti-inflammatory effects [7]

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The treatment of mouse peritoneal macrophages with an ACAT inhibitor significantly reduced 7-ketocholesterol-induced apoptosis [55]