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A hypothesized mechanism is attributed to L-cells that co-produce GLP-1 and GLP-2 at the same time, which regenerates intestinal epithelium, mediates peptide YY production, and subsequently, through appetite suppression, prolongs satiety through GLP-1 receptors and thus manages weight control [33, 47]

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When joints are inflamed or weakened, performance suffers, recovery slows, and the risk of longterm injury increases

At the core of GLP-1 therapeutics lies a remarkably simple physiological principle

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