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the endocytic adaptor Epsin binds to the ubiquitinated intracellular domain, and the resulting Clathrin-mediated endocytosis exerts a pulling force that exposes the cleavage site in the NRR [[4][4][6][5]]

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The mechanism of action remains consistent with other semaglutide formulations: it binds to GLP-1 receptors, promoting insulin release when blood glucose is elevated, and suppressing the release of glucagon, a hormone that raises blood sugar.

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