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Missing values were imputed by the predicted value from the MMRM model above, and continuous measurements were then dichotomized to binary outcomes

The intermediates from de novo lipogenesis in the liver will disrupt mitochondrial function and produce ROS, and then cause endoplasmic reticulum (ER) stress, ultimately leading to inflammation, and the broken cells releasing cytokines will cause more cell death ( The excess free fatty acids (FFAs) and de novo lipogenesis would break the homeostasis of lipid metabolism

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PLoS One 2024;19:e0306296

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