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DSS treated mice showed a huge increase (~200 fold) in IL-1 mRNA levels, whereas GLP-1-SSM caused a significant decline in the mRNA levels of IL-1 (Figure 3) in DSS + GLP-1-SSM group compared to DSS alone

On a cellular level, this molecule counteracts the phosphorylation and the inactivation of inhibitor of kappa B alpha (IB), which is responsible for the sequestration of NF-B in the cytoplasm, thus reducing the concentration of the pro-inflammatory cytokines that are primarily involved in the pathogenesis of lupus[30]

2019 update of the joint European league against rheumatism and European renal associationEuropean dialysis and transplant association (EULAR/ERAEDTA) recommendations for the management of lupus nephritis

This article explores the biological pathways, clinical evidence, and practical management strategies for heart rate changes associated with GLP-1 therapy

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Despite the extensive research on GLP-1RAs in blood glucose control and alleviation of diabetes complications, there remains a significant gap in research regarding their broader effects, especially concerning cardiovascular protection and other potential therapeutic benefits