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Previous studies show that administration of GLP-1 analogues, including exenatide (exendin-4), dulaglutide, and liraglutide in mice, rats, and nonhuman primates, suppressed outcomes related to alcohol reward, including alcohol-induced dopamine release in the nucleus accumbens, conditioned place preference for alcohol, and alcohol self-administration (6, 17)

In these patients, mean eGFR was 63 mL/min/1.73 m

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