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These latter AEs were more common in the anti-ActA and combination groups, and while these are difficult to assess in rodents and primates, we did not observe any of these in any of our preclinical in vivo studies

[32] found that exendin-4 mitigated resistance to the androgen receptor inhibitor enzalutamide in prostate cancer via the PI3K/AKT/mTOR pathway, suggesting that a combination of exendin-4 with enzalutamide may be a more effective therapeutic option for advanced prostate cancer

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