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Description

In this article, we will review incretin physiology and evidence supporting a role of the incretin hormones in the pathophysiology of hyperinsulinemic hypoglycemia, as well as results from proof-of concept studies exploring a therapeutic approach targeting the GLP-1 receptor to treat hyperinsulinemic hypoglycemia

People who continue with a program and report results are self-selected - satisfied participants are more likely to continue reporting than those with neutral or negative experiences

Clinical trials have reported gastrointestinal side effects as common adverse events with incretin-based therapies, including nausea and vomiting, which may affect patient adherence

When food reaches the small intestine and colon, specialized cells release GLP-1, which then travels to the brain to signal satiety - the feeling of being full - and to the pancreas to support appropriate insulin release in response to glucose entering your bloodstream