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CaMKK2 dysfunction may play an underlying role in this phenomenon as genetic deletion of Camkk2 in hippocampal neurons, myeloid cells and immortalized cell lines (HEK293 and HepG2) results in mitochondrial dysfunction that causes a similar switch in cellular energy generation from oxidative phosphorylation to glycolysis [43, 201, 202]

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While the candidate achieved pharmacokinetic objectives, the program was discontinued due to a single case of potential liver injury among over 1,400 trial participants

Lessons From GLP-1/Glucagon Co-Agonists in Clinical Development The first GLP-1/glucagon co-agonist to advance to human clinical trials was cotadutide (MEDI0832)

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