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This scenario leads to an increase in oxidative stress (OS), which is related to mitochondrial dysfunction, accumulations of protein and oxidated lipids, and an impairment of the antioxidant system [64,65,66,67,68,69,70]

Lipopolysaccharide-induced blood-brain barrier disruption: roles of cyclooxygenase, oxidative stress, neuroinflammation, and elements of the neurovascular unit

This high risk is largely attributable to severe cardiovascular conditions and comorbidities, such as diabetes mellitus (DM), hypertension, advanced age and immunodeficiency present at the initiation of dialysis (1-8)

The numbers around GLP-1 adoption are large enough that they deserve to be read carefully rather than cited loosely

Given that lithium activates CaMKK2, it is possible that some of the lithium-induced enhancements of mitochondrial function may be partly mediated by the CaMKK2-AMPK signalling pathway