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Isolated GCGR agonism raises blood glucose that is glucagons classical function, and it is why glucagon receptor activation has historically been considered incompatible with metabolic benefit research

Currently, prescribing guidelines for GLP-1RAs are inconsistent and largely inadequate in addressing the risk of weight regain following treatment cessation

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Together, these actions are theorised to produce a more pronounced and sustained reduction in body weight compared with single or dual receptor agonists

Also, we must take into consideration any other medical problems that can make progress slower including but not limited to polycystic ovarian syndrome, being menopausal or postmenopausal, thyroid disorders, and diabetes