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We also show that monotreme GLP-1 stimulates insulin release in cultured rodent islets, but surprisingly shows low receptor affinity and bias toward Erk signaling

Underwood et al., 2010), these structures do not give information on the receptor-bound conformation of the flexible ligand N terminus, nor on its interactions with GLP-1R

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We also investigated potential acute neuroprotective effects induced by these treatments, independently of metabolic regulation

Second, muscle loss reduces functional strength, which is particularly concerning for older adults on GLP-1 medications

Koehler et al., 2015) (Table 1)