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Compared to baseline, DIO mice assumed phylogenetically similar gut bacterial compositional changes following liraglutide and GUB09-145 treatment, characterized by discrete shifts in low-abundant species and related bacterial metabolic pathways

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In this Review, the authors describe the changes in metabolism that predispose individuals to developing atrial fibrillation (AF) and highlight the potential of available and emerging therapeutic strategies that target metabolic remodelling to treat AF.

Most side effects are gastrointestinal in nature and tend to be most noticeable during the dose escalation phase

Type 2 diabetes can be regarded as a condition of homogenous aetiology in genetically heterogenous individuals