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F., Leech, C

Various mechanisms have been theorized to describe how dual agonism, as GLP-1 RA, could directly reduce triglycerides hepatocyte storage, lipogenesis and improving hepatic glucose metabolism (96) and promoting lipolysis and fatty acid oxidation (97)

The GLP-1 receptor agonists, exenatide and liraglutide, mimic the action of endogenous GLP-1

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7 Recent data shows that GIP is responsible for a substantial portion of postprandial insulin secretion in T2DM and mild hyperglycemia in early disease

That means at least 12-16 weeks at or near the maximum tolerated dose