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Although the primary biological actions ascribed to intact GLP-1 are mediated by a single GLP-1 receptor [28], GLP-1 is rapidly cleaved by the enzyme dipeptidyl peptidase-4 (DPP-4) into GLP-1(9-36) [29], which exhibits its own biological activities (albeit not insulin secretion any more) [30,31], and thus intact GLP-1 exhibits a short half-life of one to two minutes in the circulation [32]

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Rates of diabetic ketoacidosis (DKA) and hypoglycemia were similar between groups

1,2 Beyond their metabolic effects, recent research has explored the role of GLP-1RAs in addiction regulation

2% WL with placebo in people with obesity [68]

Pimenta F, Maroco J, Ramos C, Leal I