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Chronic administration of GLP-1a, however, reduces clinic systolic BP (2 mmHg) at least when evaluated as a secondary end point in glycemia-lowering studies while simultaneously increasing heart rate

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This finding supports the role of glucose toxicity in DM-related dopaminergic dysfunction and cognitive impairment, suggesting that advanced glycation end products (AGEs) and their precursor methylglyoxal (MGO) are linked to cognitive impairment and changes in the dopaminergic system

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Therefore, the efficacy and specific benefits of GLP-1RAs in HFpEF require further investigation

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