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Exendin-4 reverses high glucose-induced endothelial progenitor cell dysfunction via SDF-1/CXCR7-AMPK/p38-MAPK/IL-6 axis

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Accordingly, this mechanism would circumvent the blocking of distinct NMDA receptors in the brainstem that might otherwise increase food intake in response to MK-801

Another recent report suggests that incretin-stimulation of insulin secretion requires incorporation of glutamate in the insulin granules
