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1994;267:E156-164
It was initially postulated to be a compensatory mechanism to the blood pressure lowering effect of GLP-1 RAs that was possibly mediated by their vasodilatory effect [9, 31] and/or increased urinary sodium excretion [32]
Detailed descriptions of the files and relevant terminology are provided in Tables S1 and S2
You can wait for market availability
5j and Extended Data Fig
They also raise concerns about a lack of follow-up care and dosage errors
