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Description
The mutation of polar hydrophobic threonine to a structural similar nonpolar hydrophobic alanine (YAP T83A ) had markedly lower levels of YAP OGlcNAcylation, and diminished interaction with EIF3H (Figure 5E,F), which led to an increased turnover of YAP (Figure 5G)
That is one reason longer phase 3 trials drew so much interest, since the ceiling had not announced itself yet
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The semaglutide onset timeline article explains the pharmacokinetics in more detail
Both marker expressions were prevented with GIP treatment [28]
