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The primary objective is to maximize gastric emptying delay and induce a massive caloric deficit
This difference in subjective effects was not explained by an increase in nausea in the GLP-1RA group
10.1152/ajpendo.00073.2012 67 KadouhH.ChedidV.HalawiH.BurtonD
In addition, early data suggests possible renal protection
Nevertheless, these data suggest that cold stress, where gut hormone receptor signaling is high, uncouples the feedback mechanisms that GLP-1R and GLP-2R govern for intestinal transit in the post-prandial state
