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Compared to a saline intraperitoneal injection, GIP release was higher following exposure to LPS in mice, and in an IL-6 and IL-1-dependent fashion, with GIP antagonism blunting the inflammatory response
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Nikolaidis LA, Elahi D, Hentosz T, Doverspike A, Huerbin R, Zourelias L, Stolarski C, Shen YT, Shannon RP
& Karamanis, D
The p38 MAPK/NF-B pathway as a potential target for DPN treatment [75], [77], [78]
