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This might indicate that the PPG neurons actually present a valuable additional drug target that could be utilised in addition to the GLP1 receptor agonists, in order to achieve increased weight loss capacity

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Regions with significantly greater c-Fos activation included the bed nuclei of the stria terminalis, paraventricular thalamus, central amygdalar nucleus (Fig

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A potential underlying mechanism involves the intracellular activation of adenylate cyclase by GLP-1RAs, leading to increased cAMP levels and subsequent activation of protein kinase A (PKA) [81]