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[22] [23] By activating GLP-1 receptors, these medications enhance insulin secretion when blood glucose levels are elevated, suppress glucagon release, slow gastric emptying, and promote satiety

The use of GLP-1 analogs was associated with improvement in the metabolic parameters of insulin resistance and lipotoxicity related to the pathogenesis of NASH

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This has peri-procedural implications due to the potential increased risk of retained gastric contents which may result in pulmonary aspiration

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