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We propose that hepatic activation of TH signaling has insulinotropic and glucose-lowering effects, as it is able to increase non-12-OH FXR-antagonistic BA levels through shaping the BA composition by repressing hepatic CYP8B1, thereby enhancing the GLP-1 production via suppressing intestinal FXR signaling

By targeting natural pathways for glucose control and fat metabolism, they promote stable energy, endurance, and sustainable weight loss

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We explored whether GLP-1(936) inhibits glucagon secretion by influencing Ca 2+ entry/exocytosis