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Longitudinal PET imaging of neuroinflammation (e.g., TSPO tracers) could serve as a biomarker for GLP-1RA efficacy in PD

Semaglutide enhances insulin secretion and suppresses glucagon release in a glucose-dependent manner, meaning these effects occur primarily when blood glucose levels are elevated

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Semaglutide-modulated pathways overlap with inflammatory signatures found in human neurodegenerative diseases, including Alzheimers disease, suggesting broad relevance for conditions involving neuroinflammation