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Furthermore, endothelial damage contributes to cardiovascular complications of COVID-19 due to ROS production induced by inflammatory cytokines, TNF- in particular

T2D remission was defined as HbA1c 40 kg/m), T2D (RCTs enrolling at least 75% of participants with diabetes), prediabetes, established CVD, HF, KOA, MASLD and OSAS

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In addition, animal studies aimed at investigating the role of inflammatory pathways in the development of ARDS and acute lung injury (ALI) due to respiratory viral infections, such as influenza virus, highlighted the role of increased activity of inflammatory pathways of the hosts immune system in disease severity and cellular damages [73]

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