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doi: 10.1007/s10072-020-04910-8

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Thus, SUs that bind SUR1 to modulate K + ATP channels independently of glucose may also allow GLP-1R agonists to bypass their inherent glucose requirement by stimulating the downstream effects ordinarily associated with increased glucose

[DOI] [PMC free article] [PubMed] [Google Scholar] 46.Dansinger M.L., Gleason J.A., Griffith J.L., Selker H.P., Schaefer E.J

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