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Normal synaptic activity can be disrupted by mitochondrial dysfunction, aberrant glutamate signaling, and toxic protein aggregates, resulting in synaptic hyperexcitability, loss of dendritic spines, and impaired plasticity

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Escalate if initial contact does not resolve the delay quickly

Structural insights into agonist-induced activation of G-protein-coupled receptors

Helps actual fat loss: Water plays a role in mobilizing and transporting fat breakdown products to the liver and kidneys for clearance

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