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Activation of these receptors produces several effects: Appetite suppression: GLP-1 agonists activate anorexigenic (appetite-suppressing) POMC/CART neurons and inhibit orexigenic (appetite-stimulating) NPY/AgRP neurons in the hypothalamus, reducing hunger signaling Enhanced satiety: Activation of brainstem GLP-1 receptors amplifies satiety signals from the gastrointestinal tract, causing patients to feel full earlier and longer after meals Reduced food reward: GLP-1 receptor activation in the mesolimbic dopamine system reduces the hedonic drive to eat, diminishing cravings for high-calorie foods (Blundell et al., Diabetes, Obesity and Metabolism , 2017) Peripheral Effects Beyond central nervous system activity, GLP-1 agonists produce peripheral effects that contribute to weight loss: Delayed gastric emptying: GLP-1 receptor activation in the stomach slows gastric motility, prolonging gastric distension and promoting earlier satiety

doi: 10.1016/j.expneurol.2013.11.001 [DOI] [PubMed] [Google Scholar] 182.Marxreiter F, Regensburger M, Winkler J

Type II diabetes is strongly correlated with obesity

Inject Slowly : Press the button and count to 6 after the dose counter shows "0"

As a growth-hormonesupporting peptide, its marketed for recovery, performance, sleep quality, and body composition

Although inulin primarily exerts its effects through gut microbiota modulation, as previously mentioned, its consumption can also directly influence human physiology