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Retatrutide (also known by its development code LY3437943) engages GLP-1, GIP, and the glucagon receptor (GCGR) through a single 39-amino-acid sequence that shares structural homology with tirzepatide but features amino acid substitutions that restore glucagon-receptor affinity

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There must be a currently unidentified physiological beneficial effect of GIPs ability to promote glucagon release in response to postprandial nutrient loads

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A calorie deficit can cause your body to shed muscle along with fat, which can slow your metabolism and make long-term success harder

Factors such as underlying health conditions, medication tolerability, and personal preferences play a significant role in determining the best treatment option