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Hyperphagia in BBS appears to result from multifactorial disruptions, whereby intrinsic defects in CCK signaling pathways and receptor function further exaggerated obesity-related resistance to satiety signals (31)

Many patients find this additional data point motivating, particularly when weight loss has reached a plateau but metabolic improvements continue

2014;306:R576R585

Proponents claim these compounds can boost fat metabolism and support weight management

GLP-1 receptor agonists, now widely prescribed for diabetes and obesity, may offer a safer option for both metabolic control and cancer prevention

This type of fat, known as visceral fat, wraps around organs and increases the risk of type 2 diabetes, fatty liver, and cardiovascular disease
