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VSMC phenotypic switching is characterized by transformation from a quiescent contractile to an activated synthetic phenotype, promoting proliferation and leading to the formation of the neointima, the pathological vascular compartment critical for the development of atherosclerosis

GLP-1 receptor agonists mimic the endogenous incretin hormone GLP-1, exerting multiple physiological effects that contribute to metabolic improvement

Patients are randomized to 2.5 to 5 years of weekly semaglutide injections or placebo as an adjunct to standard care

In obesity and T2DM: 5.0% after 16 hr, n=102

Currently, AD patients with positive tau pathology exhibit poor responsiveness to anti-A antibody treatments, which are ineffective in reducing tau levels [106]

This reduction is concerning because skeletal muscle is metabolically active tissue