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used exendin-4, an exendin-based GLP-1 analog, in hyperglycemia-induced bronchial hyperresponsiveness ex vivo, which resulted in bronchodilation that was inhibited by either cAMP-PKA antagonism or GLP-1 RA [90], a result consistent with other findings (Fig

doi: 10.1007/BF00417698

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This leads to increase in gastric volumes and putting the patients at increased risk of aspiration during sedation (107)