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Ochiai, M., Onishi, K., Yamada, T., Iida, T., & Matsuo, T
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These findings suggest that coordinated incretin and glucagon receptor activation may influence pathways involved in hepatic lipid handling and metabolic signaling [5][8]
In this review, the GLP-1/GLP-1R system implicated in diabetic endothelial dysfunction and related autophagy mechanism underlying the pathogenesis of diabetic vascular complications are briefly presented
To investigate the basis for this difference, we constructed a mouse single cell transcriptomic atlas of body and brain regions most relevant to the action of GLP-1
Consequently, cyto/chemokine expression decreased in human islets, leading to sustained islet cell viability and function
