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CCK interaction with estradiol in satiety control Cholecystokinin (CCK) is a key controller of meal-ending satiation in animals and humans ( Peripherally administered CCK reduces food intake ( Figure 1 )

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StructureActivity Relationship of GLP-1 GLP-1, after binding to its receptor, triggers a conformational change of the receptor, leading to activation of an associated G s protein by exchanging its bound GDP with a GTP

M., Rowland-Yeo, K