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However, neuronal ROS production was reduced, while astrocytic ROS levels increased, suggesting possible differential effects on mitochondrial function [85]
JACC Basic Transl Sci 2016;1:267-76.PubMedPMC 25
The mechanism is straightforward: GLP-1 RAs enhance glucose-dependent insulin secretion, and stacking that on top of glucose-independent insulin release from exogenous insulin or sulfonylureas removes the glucose-dependent safety brake
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But scientific knowledge is only half the journey
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