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Glucagon gene expression is negatively regulated by hepatocyte nuclear factor 3 beta

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Guanosine and uroguanosine, which target guanosine cyclase (GC-C) receptors in the intestine and kidney, lead to elevated intracellular cGMP concentrations, and high GC-C expression in L cells is associated with regulation of GLP-1 release (Friedlander et al., 2011)

In contrast, glucagon requires PKA and hypothetically a phosphorylation of HCN to exert a PCE (Merino et al

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