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Reports of the death of the market for continuous glucose monitors in individuals with type 2 diabetes are greatly exaggerated, Abbott Laboratories studies show

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Type 2 diabetes mellitus (T2DM) is closely linked to oxidative stressinduced mitochondrial dysfunction, disrupting the balance between fusion and fission processes and driving hepatic injury

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This is evidenced by up-regulation of glycolytic enzyme expression, increased rates of glucose uptake and consumption, as well as augmented ATP content, insulin secretion and glycolytic flux after removal of Exendin-4