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It opens the possibility of delivering remyelination-promoting peptides like TB4 or neuroprotective peptides like BPC-157 directly to MS lesions at concentrations that might not be achievable through systemic administration

However, different GLP-1R ligands can engage selective pathways to elicit a different cellular response [106]

But how they work, how well they work, and who exactly will benefit from them are harder questions to answer

L-cells are known to modulate the release of their hormonal cargo in response to the activation of a plethora of receptors capable to sense fats, carbohydrates, proteins and many other compounds

In line with these behavioral effects, GIPR agonism significantly reduced neuronal activation in the AP/NTS caused by both GLP-1 analogs and the chemotherapy agent cisplatin in shrews and rats, respectively, further supporting a role of the hindbrain in the antiemetic action of GIPR agonism ( 27 , 32 )