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Knudsens work found that liraglutide was internalized by proopiomelanocortin (POMC)/cocaine- and amphetamine-regulated transcript (CART) neurons and indirectly inhibits neuropeptide Y (NPY) and agouti-related peptide (AgRP) neurons through Gamma Amino Butyric Acid (GABA) dependent signaling

Common symptoms include mild nausea, a sense of bloating, or slight changes in bowel habits

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Taken together, the behavioral-cognitive, psychodynamic, and social-systemic lenses demonstrate that GLP-1 RA treatment is shaped not only by biological mechanisms but also by how individuals interpret bodily change and by the social environments in which treatment occurs

Further, we aimed to explore the effect of dulaglutide on the resting-state functional connectivity of homeostatic and reward-related brain regions