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We found that exendin-phe1 favored G protein signaling, whilst exendin-asp3 favored -arrestin recruitment

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It went just as well as the first

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A randomized Phase I study of the safety, tolerability, pharmacokinetics and pharmacodynamics of BI 456906, a dual glucagon receptor/glucagon-like peptide-1 receptor agonist, in healthy Japanese men with overweight/obesity