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Clinically, that translates to: More stable post-meal glucose Better appetite regulation Reduced energy crashes Improved satiety Mechanistically: GLP-1 slows gastric emptying, signals fullness centrally, supports insulin secretion, and suppresses glucagon

Many of these disorders, including obesity and auto-immune diseases, have been associated with decreased gut barrier function and consequent increased inflammation

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Scientists monitor these non-clinical models to analyze biochemical modifications in lipid substrate utilization, lipolytic enzyme activation, and downstream fatty acid oxidation cascades, providing foundational data on lipid storage mechanics at a microscopic level