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Elevated cAMP activates protein kinase A (PKA), which enhances glucose-dependent insulin gene expression and secretion while suppressing glucagon release [15,16]

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S.GormanD

If semaglutide favors sustained Gs/cAMP signaling, cAMP responses may disrupt cross-talk between cAMP and Ca 2+ pathways as it has been shown that cAMP-elevating agents (e.g., forskolin + IBMX) could suppress Ca 2+ -activated oscillatory Cl secretion in salivary acinar cells by reducing the amplitude/duration of Ca 2+ -driven pulses [74]