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In the STZ+Sm group, the ventricular edge was partially bordered by intact ependymocytes (not forming a dense layer) and vimentin-positive cells lacking the characteristic elongated basal processes of tanycytes, suggesting remodeling of the ventricular marginal layer by scar-forming, vimentine expressing astroglia or reactive ependymal changes (Fig

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Results: Soluble leptin receptor increase was 59% lower