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Several genes encoding key inflammatory mediators exhibited exceptionally high induction, such as Ccr3 (34.6-fold), Cd180 (23.8-fold), and Hck (12.5-fold)

Most recently, the Exenatide-PD3 phase III trial failed to show any advantage in motor or nonsymptoms over placebo (61), and questions remain about whether a subgroup of patients with PD may be more likely to respond to this therapy

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