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These days, demand for dairy is coming from consumers who barely registered on the radar a decade ago: users of glucagon-like peptide-1 (GLP-1) medications.
Purpose of Review This review explores the rationale and available evidence behind incretin-based therapies for metabolic dysfunction-associated steatotic disease (MASLD) and metabolic-associated steatohepatitis (MASH), focusing on glucagon-like peptide 1 (GLP-1) and glucagon (GCG) receptor dual agonists (GLP-1/GCG RAs)

These investigations help researchers examine how metabolic signaling pathways influence energy substrate utilization, lipid mobilization, and tissue-specific metabolic responses

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