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MOTS-c handles cellular energy regulation, metabolic flexibility, and mitochondrial signaling
It seems in pre-clinical models that when these L-cells are exposed to perceived toxins, such as lipopolysaccharides from certain bacteria that this triggers a surge in endogenous GLP-1 which is perhaps the bodys way of using GLP-1 as an anti-inflammatory hormone or perhaps as a brake pedal on the inflammatory response in general
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"College athletes aren't very good at disclosing what they're taking," she said
