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We and others previously demonstrated GDF8 and ActA are the two major ligands which mediate the muscle minimization actions of the ActRIIA/B receptors in non-obese mice and NHPs 22,28,30 , and we have further shown that dual blockade of GDF8 and ActA led to much larger increases in muscle mass than blocking either ligand individually, and that dual blockade matched the muscle growth actions of broader ActRIIA/B blockade 28 , similar to effects seen when using a dominant-negative type II receptor or pro-domains of GDF8 and activins 22,26,27

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How Genetic Predisposition Data Influences Long-Term Cost Efficiency Genetic variants in GLP-1 and GIP receptor pathways (such as GLP1R rs6923761 and GIPR rs1800437) may influence individual medication response rates and optimal dosing strategies