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The identification of MERCS as a structural signaling platform marks a shift toward a contactomics approach in incretin research, providing a framework to dissect how organelle miscommunication drives the progression of obesity and T2DM

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Activation of proglucagon gene transcription by protein kinase-A in a novel mouse enteroendocrine cell line

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SGLT2is and GLP-1 RAs consistently demonstrate cardiovascular and nephroprotective effects [11]

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